Subject: ICI's ex-chemical weapon insecticide causes BSE & CJD-----
Date: Fri, 02 Feb 2001 09:47:14 -0500
From: Stephen Tvedten <steve@getipm.com>
Organization: Get Set Inc. (www.getipm.com)
To: Paul Helliker <phelliker@cdpr.ca.gov>
Director, State
of California, Department of Pesticide Regulation
cc: Christine Whitman whitman.christine@epa.gov
Dear Mr. Helliker, I thought you
might like to read a very detailed article on the connection of pesticides and
mad cow disease. By a similar
process, prions in human brains can be damaged by lice lotions containing
organophosphate. Later in life, this can result in neurological diseases like
CJD and Alzheimers. The article
is located at the following url: http://eionews.addr.com/organop/organop.htm,
the article is entitled: ICI's
ex-chemical weapon insecticide causes BSE & CJD - Cover-up - Insecticide
causes Mad Cow disease - by Fintan Dunne - Research Kathy Mc Mahon - and dated
13th December 2000.
Pharmaceutical interests in the UK are ignoring new
scientific research that shows the insecticide used in the UK government's own
warble-fly campaigns triggered the UK surge of 'Mad Cow' disease.
The new work shows that prions can bond with manganese in
animal feeds or mineral licks. These manganese prions can cause the
neurological degeneration seen in BSE. And
other researchers unpublished work shows that prions in the bovine spine
--along which insecticides are applied-- can be damaged by ICI's Phosmet
organophosphate (OP) insecticide -causing the disease.
(You can clearly see how "some of"
your "registered" POISONS harm mammals by shaving the back of a
mouse's head and applying a single drop of POISON on its scalp/spine.)
By a similar process, prions in human brains can be
damaged by lice lotions containing organophosphate. Later in life, this can
result in neurological diseases like CJD and Alzheimers.
(I was at a school nurses convention where "they" were detailing the
"new" pediculicide - malathion {rhymes with prion}- "they"
were actually trying to tell the nurses that the malathion remained only on
the hair and not on the scalp! Amazing POISON, how did you or
"they" train this "registered" organophosphate to do
this?)
British scientists have led the current theory that an
infectious prion in bonemeal fed to cattle causes bovine spongiform disease (BSE).
Infectious prions are also claimed to cause new variant Creutzfeld-Jakob
Disease (CJD) in humans -from ingesting beef. But the infectious prion theory
serves to obscure a tragic chemical poisoning scandal behind the majority of
BSE cases.
Latest experiments by Cambridge University prion
specialist, David R. Brown, have shown that manganese bonds destructively with
prions. Many might be surprised to hear that organophosphates were developed
by Nazi chemists during the course World War Two, as a chemical weapon nerve
agent.
The marginalised research has devestating financial
implications for ICI. It would provide a firm basis for litigants -who could
include CJD sufferers, farmers across the world and families of the many
British farmers who committed suicide during this BSE debacle.
Phosmet organophosphate has been used at high doses in
British warble fly campaigns. In 1996, former ICI subsidiary Zeneca sold the
phosmet patent to a PO Box company in Arizona called Gowan -just one week
before the UK government admitted to a link between BSE and nvCJD.
The politically well-connected British pharmaceuticals
group, ICI has the clout to influence research into any cause other than the
infective model. Indeed no substantive alternative research has been done.
British BSE disease management and research bodies have taken decisions that
do not seem guided by spirited scientific enquiry. Mysterious prions that jump
species is the preferred research arena.
Scientist and organic farmer, Mark Purdey gave evidence
to the UK BSE inquiry, that warble fly insecticide was the cause of the
disease. The scientist
wheeled out to rubbish Purdy's evidence - Dr. David Ray, later turned out to
have been receiving funding from the insecticide manufacturer ICI.
A lobby group that includes Bayer, Monsanto, Novartis,
Pfizer, Roche and Schering-Plough was behind the effort to discredit Purdey. In
December 1999, the same David Ray was appointed to the UK Veterinary Products
Committee (VPC) -a government body that licenses animal medicines.
Purdey has been consistently denied even exploratory
funding to extend his privately supported research. Yet the Purdey/Brown
chemical poisoning model matches with the epidermiological spread of CJD
clusters in humans. It also predicts the incidence of BSE-type diseases in
animals. The accepted infectious model fits neither.
The pharmaceutical industry is all the more determined
to hide the chemical source of BSE and CJD, because a spotlight on chemicals
would expose the role the insecticides in Alzheimer's --another
neurodegenerative disease--that might lead to claims which would dwarf those
from BSE and CJD litigants. In
fact, two leading brain researchers into CJD and Alzheimers have died in
suspicious circumstances in recent years.
In the United States, the Environmental Protection Agency
is already reviewing Phosmet's safety. The Centers for Disease Control in the
US has recently conducted experiments on mice that confirm the organophosphate
risk.
Not only is the EC beef slaughter campaign futile
-because BSE disease is mostly non-infectious, but unless the underlying
chemical cause is addressed, BSE will simply reappear from chemical causes. A
new warble fly campaign is already underway in France using the
organophosphate insecticide.
Of greater concern is that some lotions for scabies
and head lice are now priming children and adults, for CJD and Alzheimers in
later life.
BONDING THE PRION
Cambridge University prion biochemist, David R. Brown is
dismissive of the science behind the infectious model of BSE. He terms it
"a very limited amount of science by a few assumed-reputable
scientists." He insists there is "no evidence an infectious agent is
present in either meat or milk."
"Simple tests on udder walls of cows --which could
easily detect an infectious prion-- have not been done, why I don't
understand." A number of researchers have found that organophosphate
(OP) in systemic warble fly insecticide can deform the prion molecule,
rendering it ineffective at buffering free radical effects in the body. Worse
still, the prion is then partial to bond with manganese and become a 'rogue'
prion. A chain reaction whereby rogue prions turn others to rogues also, can
explain the bovine spongiform disease mechanism.
Brown showed how prion protein bonds benignly with
copper, but lethally with manganese. Even natural variations in relative
environmental availability of manganese versus copper can trigger prion
degradation.
The CJD and BSE symptoms mirror 'manganese madness', an
irreversible fatal neuro-psychiatric degenerative syndrome that plagued
manganese miners in the first half of the last century
SHINING A LIGHT ON SPONGIFORM
Organic dairy farmer and peer-review-published
independent scientist, Mark Purdey, says the accepted theory of transmission
from BSE-infected cattle to human CJD -by bonemeal or meat, is dependent on a
mutant prion that has never been isolated under the scientific protocol called
Koch's postulates.
Purdey's insistence on sticking to the letter of this
scientific law earned him the condemnation of UK officialdom when he first
mooted his theory. But Purdey pointed to CJD clusters downwind of a British
Phosmet production plant to back his case.
He gave evidence to the UK Government BSE inquiry and was
supported by Conservative MP, Thessa Gorman. His views were discounted, but
his subsequent research and the new Cambridge prion work have confirmed the
alternative theory. Despite this, and the backing of a British peer, he is
denied even exploratory funding.
Speaking from his rural English Somerset farm yesterday
-as plans forge ahead for the European cattle cull, he asks:
"Why does CJD degeneration in humans begin in the retina, and
why are CJD disease clusters found in high altitude locations?"
The question is rhetorical, and Purdey has an eye-opening
answer. He argues that the prion molecule has a known natural role as a shock
adsorber of damaging energy from ultraviolet rays and other oxidizing agents.
Once this prion defense system is rendered ineffective
by organophosphates - for example in human head lice lotions, these oxidizing
effects have an unmediated impact on tissues. Eventually, UV radiation damages
the retina and oxidative stress destroys the brain tissues of CJD patients.
This theory would expect to find higher CJD incidence in mountain regions
-where UV radiation levels are elevated. That prediction holds true.
A similar but accelerated mechanism could be driving BSE.
ICI's Phosmet organophosphate warble fly insecticide - applied on the backs of
animals along the spinal column, similarly degrades prions. "Systemic
versions of the insecticide are designed to make the entire cow carcass toxic
to warble fly," explains Purdey. "Unfortunately it's toxic to prions
too - especially those prions located just millimeters from the point of
application."
The damaged prions are then ready to react with manganese
in animal feed, or manganese sprayed on land or in mineral licks - to become
the driving force of BSE neurodegeneration.
Purdey says manganese-tipped prions set off lethal chain reactions that
neurologically burn through the animal.
Chickens notoriously excrete most of the supplements fed
to them -including manganese. And their manganese-rich excreta have been
blended into cattle feed in the UK. Natural
variations in the relative environmental availability of copper and manganese
can also spur prion degeneration says Purdey.
From this research, any prudent person would conclude
there is a significant risk attaching to the use of organophosphate in humans.
Preparations for head lice and scabies are known to be overused in
practice and might be priming users for CJ disease.
Purdey believes his bias for field work is the key to his
success. He bemoans the "reductionism" of much lab-centered science.
"I have traveled the world to investigate known clusters of
spongiform disease -something mainstream researchers don't
seem remotely interested in doing."
Since first postulating an environmental
- rather than infectious- theory of spongiform diseases, Purdey has
built evidence from around the world that explains and predicts the incidence
in humans and animals: a cluster of CJD in Slovakia, Eastern Europe
- around a manganese plant; Rocky Mountain deer with Chronic Wasting
Disease (CWD), who were found to be eating pine needles rich in manganese; the
futile slaughter of sheep in Cyprus - only for BSE to reemerge within years.
"The reappearance of BSE in Cyprus obviously points
to an environmental cause," says Purdey, who is sanguine when reflecting
on the condemnation of him by mainstream scientists.
"I suppose they have mortgages and kids who need to
go to university," he muses. "Privately,
some were agreeing with me, but then they would denounce me publicly. It was
quite strange really." (What is not done for
love, is done for money.)
THE MONEY TRAIL
Critical scientists like Purdey are unlikely to
prevail. The pharma industry holds most research purse strings, and would
hardly energetically explore an avenue of research that could expose them to
litigation for causing BSE. The official theory is lavishly funded,
alternative theories rarely, if at all.
There are more explosive implications to his
- and other's latest research. Purdey says similar
organophosphate-induced protein deformation could also underlie Alzheimer's
disease. If that were true, the litigation fallout would destroy some
pharmaceutical giants, and a lot of very influential noses would be out of
joint.
Disturbingly, Purdey and other brain researchers seem to
have had an undue share of unfortunate accidents. Purdey's house was burned
down and his lawyer who was working with him on Mad Cow Disease was driven off
the road by another vehicle and subsequently died. The veterinarian on the
case also died in a car crash -locally reported as: 'Mystery Vet Death
Riddle.' (I would like to point
out that my home and then several years later my office
"accidentally" burned down.")
Dr. C. Bruton, a CJD specialist --who had just produced a
paper on a new strain of CJD--was killed in a car crash before his work was
announced to the public. Purdey speculates that Bruton might have known more
than what was revealed in his last scientific paper.
In 1996, leading Alzheimer's researcher Tsunao Saitoh, 46
and his 13 -year-old daughter were killed in La Jolla, California, in what a
Reuters report described as a "very professionally done" shooting.
What Alzheimer's Disease, Mad Cow Disease, and CJ
Disease have in common, is abnormal brain proteins and a putative link to
organophosphates. Even Gulf War syndrome among returning veterans has been
attributed, in part to the insecticide. But the sidelined scientists'
suspicions are still largely ignored.
In their favour at the moment, is a growing unease on
the part of the public. As BSE forges on and Governments panic, Science may be
out to lunch on BSE, compromised by bovine spongythinking myopathy.
Mark Purdey funds his own research, testing/ labs/travel
to cluster sites. Donations to
his research fund will help him carry on his work. Mark Purdey Research Fund,
High Barn Farm, Elworthy, Nr Taunton, Somerset TA4 3PX, UK.
Copyright © 2000 eIONews.com
Well Mr. Helliker, I believe that
most of the worry and all of the contamination in the world is caused by
people trying to make decisions before they have sufficient knowledge on which
to safely and intelligently base their decisions.
The people who make these "decision$" that are killing
us, like to hide behind what they like to call "$ound $cience.
"They" do this because they know we do not have the billion$
needed to prove their "registered" POI$ON$ are not
really "$afe". But,
as I so often point out a "$afe" POI$ON is an
oxymoron. We the people should not have to prove that your
"registered" POI$ON$ are indeed dangerous - you should
have to prove your POI$ON$ are not - AND you should be made to
pay for all of the subsequent consequences, whenever you make a
"mistake". When and if
this happens - I can virtually guarantee you, there will be no
"registered" POI$ON$! Instead of trying to stop the
use of safe and far more effective alternatives - I suggest you stop the
continued CONTAMINATION of our earth! George Patton once noted:
"Accept the challenges, so that you may feel the exhilaration of
victory." I
would like to add that: "In
an avalanche, no one snowflake thinks it's responsible."
But, Mr. Helliker, "someone" has to be responsible and we are
STILL waiting for your answer.
Respectfully, Stephen L. Tvedten
[ Steve's Additions ] [ Emphasis Added ]
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